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1.3 Objectives of this review This comprehensive review synthesizes current evidence (20192024) regarding EED-mediated reproductive toxicity in males, with particular emphasis on: Mechanistic Understanding: Elucidating the molecular pathways through which EEDs disrupt male reproductive function, including oxidative stress cascades, endocrine signaling interference, and epigenetic modifications

Figure 2 CC chemokines including CCL2, CCL3, CCL4, CCL5, CCL7, CCL13, CCL14, CCL16, CCL18, CCL19, CCL20, CCL21, and CCL25 are abnormally expressed in plasma and synovia in RA